Hamrick MW - Search Results

1

The glucocorticoid receptor in osteoprogenitors regulates bone mass and marrow fat.

Pierce JL, Ding KH, Xu J, Sharma AK, Yu K, Del Mazo Arbona N, Rodriguez-Santos Z, Bernard P, Bollag WB, Johnson MH, Hamrick MW, Begun DL, Shi XM, Isales CM, McGee-Lawrence ME. Pierce JL, et al. Among authors: hamrick mw. J Endocrinol. 2019 Jul 1:JOE-19-0230.R1. doi: 10.1530/JOE-19-0230. Online ahead of print. J Endocrinol. 2019. PMID: 31370004 Free PMC article.

2

Leptin deficiency produces contrasting phenotypes in bones of the limb and spine.

Hamrick MW, Pennington C, Newton D, Xie D, Isales C. Hamrick MW, et al. Bone. 2004 Mar;34(3):376-83. doi: 10.1016/j.bone.2003.11.020. Bone. 2004. PMID: 15003785

3

Muscle-bone interactions in dystrophin-deficient and myostatin-deficient mice.

Montgomery E, Pennington C, Isales CM, Hamrick MW. Montgomery E, et al. Among authors: hamrick mw. Anat Rec A Discov Mol Cell Evol Biol. 2005 Sep;286(1):814-22. doi: 10.1002/ar.a.20224. Anat Rec A Discov Mol Cell Evol Biol. 2005. PMID: 16078270 Free article.

4

Glucose-dependent insulinotropic polypeptide receptor knockout mice have altered bone turnover.

Xie D, Cheng H, Hamrick M, Zhong Q, Ding KH, Correa D, Williams S, Mulloy A, Bollag W, Bollag RJ, Runner RR, McPherson JC, Insogna K, Isales CM. Xie D, et al. Bone. 2005 Dec;37(6):759-69. doi: 10.1016/j.bone.2005.06.021. Epub 2005 Oct 10. Bone. 2005. PMID: 16219496

5

Disordered osteoclast formation in RAGE-deficient mouse establishes an essential role for RAGE in diabetes related bone loss.

Ding KH, Wang ZZ, Hamrick MW, Deng ZB, Zhou L, Kang B, Yan SL, She JX, Stern DM, Isales CM, Mi QS. Ding KH, et al. Among authors: hamrick mw. Biochem Biophys Res Commun. 2006 Feb 24;340(4):1091-7. doi: 10.1016/j.bbrc.2005.12.107. Epub 2005 Dec 27. Biochem Biophys Res Commun. 2006. PMID: 16403440

6

Resistance to body fat gain in 'double-muscled' mice fed a high-fat diet.

Hamrick MW, Pennington C, Webb CN, Isales CM. Hamrick MW, et al. Int J Obes (Lond). 2006 May;30(5):868-70. doi: 10.1038/sj.ijo.0803200. Int J Obes (Lond). 2006. PMID: 16404405

7

Hamrick MW, Ding KH, Pennington C, Chao YJ, Wu YD, Howard B, Immel D, Borlongan C, McNeil PL, Bollag WB, Curl WW, Yu J, Isales CM. Hamrick MW, et al. Bone. 2006 Oct;39(4):845-53. doi: 10.1016/j.bone.2006.04.011. Epub 2006 Jun 5. Bone. 2006. PMID: 16750436

8

Effects of glucose-dependent insulinotropic peptide on osteoclast function.

Zhong Q, Itokawa T, Sridhar S, Ding KH, Xie D, Kang B, Bollag WB, Bollag RJ, Hamrick M, Insogna K, Isales CM. Zhong Q, et al. Am J Physiol Endocrinol Metab. 2007 Feb;292(2):E543-8. doi: 10.1152/ajpendo.00364.2006. Epub 2006 Sep 26. Am J Physiol Endocrinol Metab. 2007. PMID: 17003233 Free article.

9

Glucose-dependent insulinotropic peptide-overexpressing transgenic mice have increased bone mass.

Xie D, Zhong Q, Ding KH, Cheng H, Williams S, Correa D, Bollag WB, Bollag RJ, Insogna K, Troiano N, Coady C, Hamrick M, Isales CM. Xie D, et al. Bone. 2007 May;40(5):1352-60. doi: 10.1016/j.bone.2007.01.007. Epub 2007 Jan 20. Bone. 2007. PMID: 17321229

10

Loss of myostatin (GDF8) function increases osteogenic differentiation of bone marrow-derived mesenchymal stem cells but the osteogenic effect is ablated with unloading.

Hamrick MW, Shi X, Zhang W, Pennington C, Thakore H, Haque M, Kang B, Isales CM, Fulzele S, Wenger KH. Hamrick MW, et al. Bone. 2007 Jun;40(6):1544-53. doi: 10.1016/j.bone.2007.02.012. Epub 2007 Feb 23. Bone. 2007. PMID: 17383950 Free PMC article.

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