Allison WT - Search Results

1

Non-Synonymous variants in premelanosome protein (PMEL) cause ocular pigment dispersion and pigmentary glaucoma.

Lahola-Chomiak AA, Footz T, Nguyen-Phuoc K, Neil GJ, Fan B, Allen KF, Greenfield DS, Parrish RK, Linkroum K, Pasquale LR, Leonhardt RM, Ritch R, Javadiyan S, Craig JE, Allison WT, Lehmann OJ, Walter MA, Wiggs JL. Lahola-Chomiak AA, et al. Among authors: allison wt. Hum Mol Genet. 2019 Apr 15;28(8):1298-1311. doi: 10.1093/hmg/ddy429. Hum Mol Genet. 2019. PMID: 30561643 Free PMC article.

2

Mutation of the bone morphogenetic protein GDF3 causes ocular and skeletal anomalies.

Ye M, Berry-Wynne KM, Asai-Coakwell M, Sundaresan P, Footz T, French CR, Abitbol M, Fleisch VC, Corbett N, Allison WT, Drummond G, Walter MA, Underhill TM, Waskiewicz AJ, Lehmann OJ. Ye M, et al. Among authors: allison wt. Hum Mol Genet. 2010 Jan 15;19(2):287-98. doi: 10.1093/hmg/ddp496. Epub 2009 Oct 28. Hum Mol Genet. 2010. PMID: 19864492

3

A complex regulatory network of transcription factors critical for ocular development and disease.

Acharya M, Huang L, Fleisch VC, Allison WT, Walter MA. Acharya M, et al. Among authors: allison wt. Hum Mol Genet. 2011 Apr 15;20(8):1610-24. doi: 10.1093/hmg/ddr038. Epub 2011 Jan 31. Hum Mol Genet. 2011. PMID: 21282189

4

Contribution of growth differentiation factor 6-dependent cell survival to early-onset retinal dystrophies.

Asai-Coakwell M, March L, Dai XH, Duval M, Lopez I, French CR, Famulski J, De Baere E, Francis PJ, Sundaresan P, Sauvé Y, Koenekoop RK, Berry FB, Allison WT, Waskiewicz AJ, Lehmann OJ. Asai-Coakwell M, et al. Among authors: allison wt. Hum Mol Genet. 2013 Apr 1;22(7):1432-42. doi: 10.1093/hmg/dds560. Epub 2013 Jan 9. Hum Mol Genet. 2013. PMID: 23307924

5

Longitudinal fluorescent observation of retinal degeneration and regeneration in zebrafish using fundus lens imaging.

Duval MG, Chung H, Lehmann OJ, Allison WT. Duval MG, et al. Among authors: allison wt. Mol Vis. 2013 May 23;19:1082-95. Print 2013. Mol Vis. 2013. PMID: 23734077 Free PMC article.

6

Reduced Abundance and Subverted Functions of Proteins in Prion-Like Diseases: Gained Functions Fascinate but Lost Functions Affect Aetiology.

Allison WT, DuVal MG, Nguyen-Phuoc K, Leighton PLA. Allison WT, et al. Int J Mol Sci. 2017 Oct 24;18(10):2223. doi: 10.3390/ijms18102223. Int J Mol Sci. 2017. PMID: 29064456 Free PMC article. Review.

7

Prion gene paralogs are dispensable for early zebrafish development and have nonadditive roles in seizure susceptibility.

Leighton PLA, Kanyo R, Neil GJ, Pollock NM, Allison WT. Leighton PLA, et al. Among authors: allison wt. J Biol Chem. 2018 Aug 10;293(32):12576-12592. doi: 10.1074/jbc.RA117.001171. Epub 2018 Jun 14. J Biol Chem. 2018. PMID: 29903907 Free PMC article.

8

Amyloid-β precursor protein mutant zebrafish exhibit seizure susceptibility that depends on prion protein.

Kanyo R, Leighton PLA, Neil GJ, Locskai LF, Allison WT. Kanyo R, et al. Among authors: allison wt. Exp Neurol. 2020 Jun;328:113283. doi: 10.1016/j.expneurol.2020.113283. Epub 2020 Mar 9. Exp Neurol. 2020. PMID: 32165257

9

Nrl Is Dispensable for Specification of Rod Photoreceptors in Adult Zebrafish Despite Its Deeply Conserved Requirement Earlier in Ontogeny.

Oel AP, Neil GJ, Dong EM, Balay SD, Collett K, Allison WT. Oel AP, et al. Among authors: allison wt. iScience. 2020 Nov 15;23(12):101805. doi: 10.1016/j.isci.2020.101805. eCollection 2020 Dec 18. iScience. 2020. PMID: 33299975 Free PMC article.

10

Transcriptomic analysis of zebrafish prion protein mutants supports conserved cross-species function of the cellular prion protein.

Pollock NM, Leighton P, Neil G, Allison WT. Pollock NM, et al. Among authors: allison wt. Prion. 2021 Dec;15(1):70-81. doi: 10.1080/19336896.2021.1924557. Prion. 2021. PMID: 34139950 Free PMC article.

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