Kalchschmidt J - Search Results

1

EBV persistence without its EBNA3A and 3C oncogenes in vivo.

Murer A, McHugh D, Caduff N, Kalchschmidt J, Barros M, Zbinden A, Capaul R, Niedobitek G, Allday M, Chijioke O, Münz C. Murer A, et al. Among authors: kalchschmidt j. PLoS Pathog. 2018 Apr 30;14(4):e1007039. doi: 10.1371/journal.ppat.1007039. eCollection 2018 Apr. PLoS Pathog. 2018. PMID: 29709016 Free PMC article.

2

Epstein-Barr Virus Proteins EBNA3A and EBNA3C Together Induce Expression of the Oncogenic MicroRNA Cluster miR-221/miR-222 and Ablate Expression of Its Target p57KIP2.

Bazot Q, Paschos K, Skalska L, Kalchschmidt JS, Parker GA, Allday MJ. Bazot Q, et al. Among authors: kalchschmidt js. PLoS Pathog. 2015 Jul 8;11(7):e1005031. doi: 10.1371/journal.ppat.1005031. eCollection 2015 Jul. PLoS Pathog. 2015. PMID: 26153983 Free PMC article.

3

Plasmacytoid dendritic cells respond to Epstein-Barr virus infection with a distinct type I interferon subtype profile.

Gujer C, Murer A, Müller A, Vanoaica D, Sutter K, Jacque E, Fournier N, Kalchschmidt J, Zbinden A, Capaul R, Dzionek A, Mondon P, Dittmer U, Münz C. Gujer C, et al. Among authors: kalchschmidt j. Blood Adv. 2019 Apr 9;3(7):1129-1144. doi: 10.1182/bloodadvances.2018025536. Blood Adv. 2019. PMID: 30952679 Free PMC article.

4

EBNA3C Directs Recruitment of RBPJ (CBF1) to Chromatin during the Process of Gene Repression in EBV Infected B Cells.

Kalchschmidt JS, Gillman AC, Paschos K, Bazot Q, Kempkes B, Allday MJ. Kalchschmidt JS, et al. PLoS Pathog. 2016 Jan 11;12(1):e1005383. doi: 10.1371/journal.ppat.1005383. eCollection 2016 Jan. PLoS Pathog. 2016. PMID: 26751214 Free PMC article.

5

Epstein-Barr virus nuclear protein EBNA3C directly induces expression of AID and somatic mutations in B cells.

Kalchschmidt JS, Bashford-Rogers R, Paschos K, Gillman AC, Styles CT, Kellam P, Allday MJ. Kalchschmidt JS, et al. J Exp Med. 2016 May 30;213(6):921-8. doi: 10.1084/jem.20160120. Epub 2016 May 23. J Exp Med. 2016. PMID: 27217538 Free PMC article.

6

In vivo time-lapse imaging shows diverse niche engagement by quiescent and naturally activated hematopoietic stem cells.

Rashidi NM, Scott MK, Scherf N, Krinner A, Kalchschmidt JS, Gounaris K, Selkirk ME, Roeder I, Lo Celso C. Rashidi NM, et al. Among authors: kalchschmidt js. Blood. 2014 Jul 3;124(1):79-83. doi: 10.1182/blood-2013-10-534859. Epub 2014 May 21. Blood. 2014. PMID: 24850759 Free PMC article.

7

A Pliable Mediator Acts as a Functional Rather Than an Architectural Bridge between Promoters and Enhancers.

El Khattabi L, Zhao H, Kalchschmidt J, Young N, Jung S, Van Blerkom P, Kieffer-Kwon P, Kieffer-Kwon KR, Park S, Wang X, Krebs J, Tripathi S, Sakabe N, Sobreira DR, Huang SC, Rao SSP, Pruett N, Chauss D, Sadler E, Lopez A, Nóbrega MA, Aiden EL, Asturias FJ, Casellas R. El Khattabi L, et al. Among authors: kalchschmidt j. Cell. 2019 Aug 22;178(5):1145-1158.e20. doi: 10.1016/j.cell.2019.07.011. Epub 2019 Aug 8. Cell. 2019. PMID: 31402173 Free PMC article.

8

Structure of mammalian Mediator complex reveals Tail module architecture and interaction with a conserved core.

Zhao H, Young N, Kalchschmidt J, Lieberman J, El Khattabi L, Casellas R, Asturias FJ. Zhao H, et al. Among authors: kalchschmidt j. Nat Commun. 2021 Mar 1;12(1):1355. doi: 10.1038/s41467-021-21601-w. Nat Commun. 2021. PMID: 33649303 Free PMC article.

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