Segal BM - Search Results

1

CXCL13 promotes isotype-switched B cell accumulation to the central nervous system during viral encephalomyelitis.

Phares TW, DiSano KD, Stohlman SA, Segal BM, Bergmann CC. Phares TW, et al. Among authors: segal bm. Brain Behav Immun. 2016 May;54:128-139. doi: 10.1016/j.bbi.2016.01.016. Epub 2016 Jan 18. Brain Behav Immun. 2016. PMID: 26795429 Free PMC article.

2

The lymphoid chemokine, CXCL13, is dispensable for the initial recruitment of B cells to the acutely inflamed central nervous system.

Rainey-Barger EK, Rumble JM, Lalor SJ, Esen N, Segal BM, Irani DN. Rainey-Barger EK, et al. Among authors: segal bm. Brain Behav Immun. 2011 Jul;25(5):922-31. doi: 10.1016/j.bbi.2010.10.002. Epub 2010 Oct 8. Brain Behav Immun. 2011. PMID: 20933590 Free PMC article.

3

IL-23 modulated myelin-specific T cells induce EAE via an IFNγ driven, IL-17 independent pathway.

Kroenke MA, Segal BM. Kroenke MA, et al. Among authors: segal bm. Brain Behav Immun. 2011 Jul;25(5):932-7. doi: 10.1016/j.bbi.2010.10.001. Epub 2010 Oct 15. Brain Behav Immun. 2011. PMID: 20951792 Free PMC article.

4

Notch signaling regulates T cell accumulation and function in the central nervous system during experimental autoimmune encephalomyelitis.

Sandy AR, Stoolman J, Malott K, Pongtornpipat P, Segal BM, Maillard I. Sandy AR, et al. Among authors: segal bm. J Immunol. 2013 Aug 15;191(4):1606-13. doi: 10.4049/jimmunol.1301116. Epub 2013 Jul 3. J Immunol. 2013. PMID: 23825310 Free PMC article.

5

T-bet promotes the accumulation of encephalitogenic Th17 cells in the CNS.

Grifka-Walk HM, Segal BM. Grifka-Walk HM, et al. Among authors: segal bm. J Neuroimmunol. 2017 Mar 15;304:35-39. doi: 10.1016/j.jneuroim.2016.05.007. Epub 2016 May 24. J Neuroimmunol. 2017. PMID: 27242075 Free PMC article.

6

GM-CSF Promotes Chronic Disability in Experimental Autoimmune Encephalomyelitis by Altering the Composition of Central Nervous System-Infiltrating Cells, but Is Dispensable for Disease Induction.

Duncker PC, Stoolman JS, Huber AK, Segal BM. Duncker PC, et al. Among authors: segal bm. J Immunol. 2018 Feb 1;200(3):966-973. doi: 10.4049/jimmunol.1701484. Epub 2017 Dec 29. J Immunol. 2018. PMID: 29288202 Free PMC article.

7

Getting to the crux of the matter: IL-23 and Th17 cell accumulation in the CNS.

Segal BM. Segal BM. Eur J Immunol. 2009 Jul;39(7):1713-5. doi: 10.1002/eji.200939675. Eur J Immunol. 2009. PMID: 19582738 Free PMC article.

8

Th17 cells in autoimmune demyelinating disease.

Segal BM. Segal BM. Semin Immunopathol. 2010 Mar;32(1):71-7. doi: 10.1007/s00281-009-0186-z. Epub 2010 Feb 27. Semin Immunopathol. 2010. PMID: 20195867 Free PMC article. Review.

9

Lymphoid chemokines in the CNS.

Lalor SJ, Segal BM. Lalor SJ, et al. Among authors: segal bm. J Neuroimmunol. 2010 Jul 27;224(1-2):56-61. doi: 10.1016/j.jneuroim.2010.05.017. Epub 2010 Jun 1. J Neuroimmunol. 2010. PMID: 20605642 Free PMC article. Review.

10

The unwavering commitment of regulatory T cells in the suppression of autoimmune encephalomyelitis: another aspect of immune privilege in the CNS.

Segal BM. Segal BM. Eur J Immunol. 2012 May;42(5):1102-5. doi: 10.1002/eji.201242567. Eur J Immunol. 2012. PMID: 22539285 Free PMC article.

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