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CO₂directly modulates connexin 26 by formation of carbamate bridges between subunits.
Elife. 2013 Nov 12;2:e01213. doi: 10.7554/eLife.01213.
Elife. 2013.
PMID: 24220509
Free PMC article.
Connexin26 hemichannels with a mutation that causes KID syndrome in humans lack sensitivity to CO2.
Meigh L, Hussain N, Mulkey DK, Dale N.
Meigh L, et al.
Elife. 2014 Nov 25;3:e04249. doi: 10.7554/eLife.04249.
Elife. 2014.
PMID: 25422938
Free PMC article.
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Opposing modulation of Cx26 gap junctions and hemichannels by CO2.
Nijjar S, Maddison D, Meigh L, de Wolf E, Rodgers T, Cann MJ, Dale N.
Nijjar S, et al. Among authors: meigh l.
J Physiol. 2021 Jan;599(1):103-118. doi: 10.1113/JP280747. Epub 2020 Oct 22.
J Physiol. 2021.
PMID: 33022747
Free article.
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Rational design of new NO and redox sensitivity into connexin26 hemichannels.
Meigh L, Cook D, Zhang J, Dale N.
Meigh L, et al.
Open Biol. 2015 Feb;5(2):140208. doi: 10.1098/rsob.140208.
Open Biol. 2015.
PMID: 25673329
Free PMC article.
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Connexin26 mediates CO2-dependent regulation of breathing via glial cells of the medulla oblongata.
van de Wiel J, Meigh L, Bhandare A, Cook J, Nijjar S, Huckstepp R, Dale N.
van de Wiel J, et al. Among authors: meigh l.
Commun Biol. 2020 Sep 21;3(1):521. doi: 10.1038/s42003-020-01248-x.
Commun Biol. 2020.
PMID: 32958814
Free PMC article.
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CO2 carbamylation of proteins as a mechanism in physiology.
Meigh L.
Meigh L.
Biochem Soc Trans. 2015 Jun;43(3):460-4. doi: 10.1042/BST20150026.
Biochem Soc Trans. 2015.
PMID: 26009191
Review.
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