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Metabolic engineering of the iodine content in Arabidopsis.
Landini M, Gonzali S, Kiferle C, Tonacchera M, Agretti P, Dimida A, Vitti P, Alpi A, Pinchera A, Perata P. Landini M, et al. Among authors: vitti p. Sci Rep. 2012;2:338. doi: 10.1038/srep00338. Epub 2012 Mar 27. Sci Rep. 2012. PMID: 22468225 Free PMC article.
Iodine fortification of vegetables improves human iodine nutrition: in vivo evidence for a new model of iodine prophylaxis.
Tonacchera M, Dimida A, De Servi M, Frigeri M, Ferrarini E, De Marco G, Grasso L, Agretti P, Piaggi P, Aghini-Lombardi F, Perata P, Pinchera A, Vitti P. Tonacchera M, et al. Among authors: vitti p. J Clin Endocrinol Metab. 2013 Apr;98(4):E694-7. doi: 10.1210/jc.2012-3509. Epub 2013 Mar 12. J Clin Endocrinol Metab. 2013. PMID: 23482609 Clinical Trial.
Assessment of nodular goitre.
Tonacchera M, Pinchera A, Vitti P. Tonacchera M, et al. Among authors: vitti p. Best Pract Res Clin Endocrinol Metab. 2010 Feb;24(1):51-61. doi: 10.1016/j.beem.2009.08.008. Best Pract Res Clin Endocrinol Metab. 2010. PMID: 20172470 Review.
Benign nonfunctioning thyroid adenomas are characterized by a defective targeting to cell membrane or a reduced expression of the sodium iodide symporter protein.
Tonacchera M, Viacava P, Agretti P, de Marco G, Perri A, di Cosmo C, de Servi M, Miccoli P, Lippi F, Naccarato AG, Pinchera A, Chiovato L, Vitti P. Tonacchera M, et al. Among authors: vitti p. J Clin Endocrinol Metab. 2002 Jan;87(1):352-7. doi: 10.1210/jcem.87.1.8173. J Clin Endocrinol Metab. 2002. PMID: 11788674
The sodium-iodide symporter protein is always present at a low expression and confined to the cell membrane in nonfunctioning nonadenomatous nodules of toxic nodular goitre.
Tonacchera M, Viacava P, Fanelli G, Agretti P, De Marco G, De Servi M, Di Cosmo C, Chiovato L, Pinchera A, Vitti P. Tonacchera M, et al. Among authors: vitti p. Clin Endocrinol (Oxf). 2004 Jul;61(1):40-5. doi: 10.1111/j.1365-2265.2004.02047.x. Clin Endocrinol (Oxf). 2004. PMID: 15212643
Ras homolog enriched in striatum inhibits the functional activity of wild type thyrotropin, follicle-stimulating hormone, luteinizing hormone receptors and activating thyrotropin receptor mutations by altering their expression in COS-7 cells.
Agretti P, De Marco G, Pinchera A, Vitti P, Bernal J, Tonacchera M. Agretti P, et al. Among authors: vitti p. J Endocrinol Invest. 2007 Apr;30(4):279-84. doi: 10.1007/BF03346294. J Endocrinol Invest. 2007. PMID: 17556863
371 results