Fanzani A - Search Results

1

Caveolin 1 is a marker of poor differentiation in Rhabdomyosarcoma.

Rossi S, Poliani PL, Cominelli M, Bozzato A, Vescovi R, Monti E, Fanzani A. Rossi S, et al. Among authors: fanzani a. Eur J Cancer. 2011 Mar;47(5):761-72. doi: 10.1016/j.ejca.2010.10.018. Epub 2010 Nov 22. Eur J Cancer. 2011. PMID: 21106364

2

Hypertrophy and atrophy inversely regulate Caveolin-3 expression in myoblasts.

Fanzani A, Musarò A, Stoppani E, Giuliani R, Colombo F, Preti A, Marchesini S. Fanzani A, et al. Biochem Biophys Res Commun. 2007 May 25;357(1):314-8. doi: 10.1016/j.bbrc.2007.03.148. Epub 2007 Apr 2. Biochem Biophys Res Commun. 2007. PMID: 17418092

3

Phenotypic behavior of C2C12 myoblasts upon expression of the dystrophy-related caveolin-3 P104L and TFT mutants.

Fanzani A, Stoppani E, Gualandi L, Giuliani R, Galbiati F, Rossi S, Fra A, Preti A, Marchesini S. Fanzani A, et al. FEBS Lett. 2007 Oct 30;581(26):5099-104. doi: 10.1016/j.febslet.2007.09.055. Epub 2007 Oct 4. FEBS Lett. 2007. PMID: 17935719 Free article.

4

The enzymatic activity of sialidase Neu2 is inversely regulated during in vitro myoblast hypertrophy and atrophy.

Fanzani A, Giuliani R, Colombo F, Rossi S, Stoppani E, Martinet W, Preti A, Marchesini S. Fanzani A, et al. Biochem Biophys Res Commun. 2008 May 30;370(2):376-81. doi: 10.1016/j.bbrc.2008.03.111. Epub 2008 Mar 31. Biochem Biophys Res Commun. 2008. PMID: 18384747

5

The cytosolic sialidase Neu2 is degraded by autophagy during myoblast atrophy.

Rossi S, Stoppani E, Martinet W, Bonetto A, Costelli P, Giuliani R, Colombo F, Preti A, Marchesini S, Fanzani A. Rossi S, et al. Among authors: fanzani a. Biochim Biophys Acta. 2009 Aug;1790(8):817-28. doi: 10.1016/j.bbagen.2009.04.006. Epub 2009 Apr 14. Biochim Biophys Acta. 2009. PMID: 19371771

6

Defective myogenic differentiation of human rhabdomyosarcoma cells is characterized by sialidase Neu2 loss of expression.

Stoppani E, Rossi S, Marchesini S, Preti A, Fanzani A. Stoppani E, et al. Among authors: fanzani a. Cell Biol Int. 2009 Sep;33(9):1020-5. doi: 10.1016/j.cellbi.2009.06.005. Epub 2009 Jun 12. Cell Biol Int. 2009. PMID: 19524683

7

L6E9 myoblasts are deficient of myostatin and additional TGF-beta members are candidates to developmentally control their fiber formation.

Rossi S, Stoppani E, Gobbo M, Caroli A, Fanzani A. Rossi S, et al. Among authors: fanzani a. J Biomed Biotechnol. 2010;2010:326909. doi: 10.1155/2010/326909. Epub 2010 Apr 13. J Biomed Biotechnol. 2010. PMID: 20396675 Free PMC article.

8

Cisplatin triggers atrophy of skeletal C2C12 myotubes via impairment of Akt signalling pathway and subsequent increment activity of proteasome and autophagy systems.

Fanzani A, Zanola A, Rovetta F, Rossi S, Aleo MF. Fanzani A, et al. Toxicol Appl Pharmacol. 2011 Feb 1;250(3):312-21. doi: 10.1016/j.taap.2010.11.003. Epub 2010 Nov 11. Toxicol Appl Pharmacol. 2011. PMID: 21074548

9

Point mutated caveolin-3 form (P104L) impairs myoblast differentiation via Akt and p38 signalling reduction, leading to an immature cell signature.

Stoppani E, Rossi S, Meacci E, Penna F, Costelli P, Bellucci A, Faggi F, Maiolo D, Monti E, Fanzani A. Stoppani E, et al. Among authors: fanzani a. Biochim Biophys Acta. 2011 Apr;1812(4):468-79. doi: 10.1016/j.bbadis.2010.12.005. Epub 2010 Dec 21. Biochim Biophys Acta. 2011. PMID: 21182936 Free article.

10

Differentiation of human rhabdomyosarcoma RD cells is regulated by reciprocal, functional interactions between myostatin, p38 and extracellular regulated kinase signalling pathways.

Rossi S, Stoppani E, Puri PL, Fanzani A. Rossi S, et al. Among authors: fanzani a. Eur J Cancer. 2011 May;47(7):1095-105. doi: 10.1016/j.ejca.2010.12.010. Epub 2011 Jan 25. Eur J Cancer. 2011. PMID: 21273059

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