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Postinfarction treatment with an adenoviral vector expressing hepatocyte growth factor relieves chronic left ventricular remodeling and dysfunction in mice.
Li Y, Takemura G, Kosai K, Yuge K, Nagano S, Esaki M, Goto K, Takahashi T, Hayakawa K, Koda M, Kawase Y, Maruyama R, Okada H, Minatoguchi S, Mizuguchi H, Fujiwara T, Fujiwara H. Li Y, et al. Among authors: takemura g. Circulation. 2003 May 20;107(19):2499-506. doi: 10.1161/01.CIR.0000065579.19126.B8. Epub 2003 Apr 14. Circulation. 2003. PMID: 12695295
Inhibition of granulation tissue cell apoptosis during the subacute stage of myocardial infarction improves cardiac remodeling and dysfunction at the chronic stage.
Hayakawa K, Takemura G, Kanoh M, Li Y, Koda M, Kawase Y, Maruyama R, Okada H, Minatoguchi S, Fujiwara T, Fujiwara H. Hayakawa K, et al. Among authors: takemura g. Circulation. 2003 Jul 8;108(1):104-9. doi: 10.1161/01.CIR.0000074225.62168.68. Epub 2003 Jun 23. Circulation. 2003. PMID: 12821555
Acceleration of the healing process and myocardial regeneration may be important as a mechanism of improvement of cardiac function and remodeling by postinfarction granulocyte colony-stimulating factor treatment.
Minatoguchi S, Takemura G, Chen XH, Wang N, Uno Y, Koda M, Arai M, Misao Y, Lu C, Suzuki K, Goto K, Komada A, Takahashi T, Kosai K, Fujiwara T, Fujiwara H. Minatoguchi S, et al. Among authors: takemura g. Circulation. 2004 Jun 1;109(21):2572-80. doi: 10.1161/01.CIR.0000129770.93985.3E. Epub 2004 May 3. Circulation. 2004. PMID: 15123535
Alpha1-receptor or adenosine A1-receptor dependent pathway alone is not sufficient but summation of these pathways is required to achieve an ischaemic preconditioning effect in rabbits.
Baba K, Minatoguchi S, Zhang C, Kariya T, Uno Y, Kawai T, Takahashi M, Takemura G, Fujiwara H. Baba K, et al. Among authors: takemura g. Clin Exp Pharmacol Physiol. 2005 Apr;32(4):263-8. doi: 10.1111/j.0305-1870.2005.04185.x. Clin Exp Pharmacol Physiol. 2005. PMID: 15810989
266 results