Plasmodium falciparum: soluble thrombospondin increases cytoadherence of parasitized erythrocytes to human microvascular endothelium under shear flow conditions

Exp Parasitol. 1997 Sep;87(1):69-72. doi: 10.1006/expr.1997.4186.

Authors

J P Siano¹, K K Grady, P Millet, R A Swerlick, T M Wick

Affiliation

¹ School of Chemical Engineering, Georgia Institute of Technology, Atlanta 30332-0100, USA.

PMID: 9287960
DOI: 10.1006/expr.1997.4186

No abstract available

MeSH terms

Analysis of Variance
Animals
CD36 Antigens / metabolism
Cell Adhesion
Cell Adhesion Molecules / blood*
Cell Adhesion Molecules / chemistry
Cell Adhesion Molecules / physiology
Cells, Cultured
Culture Media, Serum-Free
Endothelium, Vascular / cytology
Endothelium, Vascular / metabolism*
Erythrocytes / metabolism
Erythrocytes / parasitology*
Humans
Malaria, Falciparum / blood*
Malaria, Falciparum / etiology
Membrane Glycoproteins / blood*
Membrane Glycoproteins / chemistry
Membrane Glycoproteins / physiology
Microcirculation
Plasmodium falciparum / metabolism*
Solubility
Thrombospondins

Substances

CD36 Antigens
Cell Adhesion Molecules
Culture Media, Serum-Free
Membrane Glycoproteins
Thrombospondins