Thrombus formation in the microvessels and endocardium was suggestive of endothelial cell damage, myocardial ischemia, and a decreased coronary flow reserve. Sustained pulmonary hypertension due to thrombosis worsened the biventricular dysfunction.
Keywords: biventricular systolic dysfunction; mitral valve replacement; mural thrombi; myocardial infarction with non‐obstructive coronary arteries; non‐ischemic dilated cardiomyopathy; stuck valve.
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