Age-associated transcriptional stress due to accelerated elongation and increased stalling of RNAPII

Nat Genet. 2023 Dec;55(12):2011-2012. doi: 10.1038/s41588-023-01601-w.

Antonios Papadakis¹, Akos Gyenis^{2

3}, Joris Pothof², Jan H J Hoeijmakers^{2

3

4}, Argyris Papantonis⁵, Andreas Beyer^{6

7

8}

¹ Cluster of Excellence on Cellular Stress Responses in Aging-associated Diseases (CECAD), University of Cologne, Cologne, Germany.
² Department of Molecular Genetics, Erasmus MC Cancer Institute, Erasmus University Medical Center, Rotterdam, the Netherlands.
³ University of Cologne, Faculty of Medicine, Cluster of Excellence on Cellular Stress Responses in Aging-associated Diseases (CECAD), Institute for Genome Stability in Ageing and Disease, Cologne, Germany.
⁴ Princess Maxima Center for Pediatric Oncology, Oncode Institute, Utrecht, The Netherlands.
⁵ Institute of Pathology, University Medical Centre Göttingen, Göttingen, Germany.
⁶ Cluster of Excellence on Cellular Stress Responses in Aging-associated Diseases (CECAD), University of Cologne, Cologne, Germany. andreas.beyer@uni-koeln.de.
⁷ Center for Molecular Medicine Cologne (CMMC), University of Cologne, Faculty of Medicine and University Hospital Cologne, Cologne, Germany. andreas.beyer@uni-koeln.de.
⁸ Institute for Genetics, Faculty of Mathematics and Natural Sciences, University of Cologne, Cologne, Germany. andreas.beyer@uni-koeln.de.

No abstract available

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