On target inhibition of vascular smooth muscle cell phenotypic transition underpins TNF-OXPHOS-AP-1 as a promising avenue for anti-remodelling interventions in aortic dissection and rupture

Eur Heart J. 2024 Jan 27;45(4):306-308. doi: 10.1093/eurheartj/ehad679.
No abstract available

Publication types

  • Editorial
  • Comment

MeSH terms

  • Aortic Dissection* / surgery
  • Cells, Cultured
  • Humans
  • Muscle, Smooth, Vascular / physiology
  • Myocytes, Smooth Muscle / physiology
  • Transcription Factor AP-1
  • Vascular Diseases*

Substances

  • Transcription Factor AP-1