Rewiring of catecholamine-induced calcium signalling is an early event in non-alcoholic fatty liver disease

J Physiol. 2023 Apr;601(8):1317-1318. doi: 10.1113/JP284526. Epub 2023 Mar 29.

Authors

Shane C Wright^{1

2

3}, Volker M Lauschke^{1

4

5}

Affiliations

¹ Department of Physiology & Pharmacology, Karolinska Institutet, Stockholm, Sweden.
² Department of Biochemistry and Molecular Medicine, Université de Montréal, Montréal, QC, Canada.
³ Institute for Research in Immunology and Cancer, Université de Montréal, Montréal, QC, Canada.
⁴ Dr Margarete Fischer-Bosch Institute of Clinical Pharmacology, Stuttgart, Germany.
⁵ University of Tübingen, Tübingen, Germany.

PMID: 36967613
DOI: 10.1113/JP284526

No abstract available

Keywords: G protein-coupled receptor; calcium oscillations; inositol 1,4,5-trisphosphate; metabolic disease; non-alcoholic fatty liver disease.

Publication types

Research Support, Non-U.S. Gov't

MeSH terms

Calcium / metabolism
Calcium Signaling
Catecholamines / metabolism
Humans
Inositol 1,4,5-Trisphosphate / metabolism
Inositol 1,4,5-Trisphosphate Receptors / metabolism
Liver / metabolism
Non-alcoholic Fatty Liver Disease* / metabolism

Substances

Calcium
Catecholamines
Inositol 1,4,5-Trisphosphate Receptors
Inositol 1,4,5-Trisphosphate