Abstract
Posttranslational modifications contribute to the pathology of methylmalonic acidemia and may be targetable via an acylation-resistant sirtuin (Head et al., this issue).
Publication types
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Review
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Research Support, Non-U.S. Gov't
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Comment
MeSH terms
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Acylation
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Amino Acid Metabolism, Inborn Errors*
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Humans
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Lysine* / metabolism
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Protein Processing, Post-Translational