The interaction between SARS-CoV-2 and ACE2 may have consequences for skeletal muscle viral susceptibility and myopathies

J Appl Physiol (1985). 2020 Oct 1;129(4):864-867. doi: 10.1152/japplphysiol.00321.2020. Epub 2020 Jul 16.
No abstract available

Keywords: COVID-19; SARS-CoV-2; muscular dystrophy; myopathy; respiration; sarcopenia; skeletal muscle.

Publication types

  • Review

MeSH terms

  • Angiotensin-Converting Enzyme 2
  • Betacoronavirus*
  • COVID-19
  • Coronavirus Infections / pathology
  • Coronavirus Infections / virology*
  • Disease Susceptibility
  • Humans
  • Muscle, Skeletal / metabolism
  • Muscle, Skeletal / pathology
  • Muscle, Skeletal / virology*
  • Muscular Diseases / metabolism
  • Muscular Diseases / pathology
  • Muscular Diseases / virology*
  • Pandemics
  • Peptidyl-Dipeptidase A / metabolism*
  • Pneumonia, Viral / pathology
  • Pneumonia, Viral / virology*
  • Respiration Disorders / metabolism
  • Respiration Disorders / pathology
  • Respiration Disorders / virology
  • Respiratory Muscles / metabolism
  • Respiratory Muscles / pathology
  • Respiratory Muscles / virology
  • SARS-CoV-2

Substances

  • Peptidyl-Dipeptidase A
  • ACE2 protein, human
  • Angiotensin-Converting Enzyme 2