The Interaction of Person-Affect-Cognition-Execution (I-PACE) model for addictive behaviors: Update, generalization to addictive behaviors beyond internet-use disorders, and specification of the process character of addictive behaviors

Neurosci Biobehav Rev. 2019 Sep:104:1-10. doi: 10.1016/j.neubiorev.2019.06.032. Epub 2019 Jun 24.

Abstract

We propose an updated version of the Interaction of Person-Affect-Cognition-Execution (I-PACE) model, which we argue to be valid for several types of addictive behaviors, such as gambling, gaming, buying-shopping, and compulsive sexual behavior disorders. Based on recent empirical findings and theoretical considerations, we argue that addictive behaviors develop as a consequence of the interactions between predisposing variables, affective and cognitive responses to specific stimuli, and executive functions, such as inhibitory control and decision-making. In the process of addictive behaviors, the associations between cue-reactivity/craving and diminished inhibitory control contribute to the development of habitual behaviors. An imbalance between structures of fronto-striatal circuits, particularly between ventral striatum, amygdala, and dorsolateral prefrontal areas, may be particularly relevant to early stages and the dorsal striatum to later stages of addictive processes. The I-PACE model may provide a theoretical foundation for future studies on addictive behaviors and clinical practice. Future studies should investigate common and unique mechanisms involved in addictive, obsessive-compulsive-related, impulse-control, and substance-use disorders.

Keywords: Behavioral addictions; Buying-shopping disorder; Cue-reactivity; Gambling disorder; Gaming disorder; Inhibitory control; Problematic pornography use.

Publication types

  • Research Support, Non-U.S. Gov't
  • Review

MeSH terms

  • Amygdala / physiopathology*
  • Behavior, Addictive / physiopathology*
  • Decision Making / physiology*
  • Executive Function / physiology*
  • Humans
  • Inhibition, Psychological*
  • Models, Theoretical*
  • Prefrontal Cortex / physiopathology*
  • Ventral Striatum / physiopathology*