Heterozygous Deletion of EphrinA5 Does Not Improve Functional Recovery After Experimental Stroke

Stroke. 2019 Apr;50(4):e101. doi: 10.1161/STROKEAHA.118.023857.

Authors

Antina de Boer^{1

2}, Annet Storm^{1

2}, Laura Rué^{1

2}, Lindsay Poppe^{1

2}, Wim Robberecht^{1

2

3}, Robin Lemmens^{1

2

3}

Affiliations

¹ From the KU Leuven-University of Leuven, Department of Neurosciences, Experimental Neurology, and Leuven Brain Institute (LBI), Belgium (A.d.B., A.S., L.R., L.P., W.R., R.L.).
² VIB, Center for Brain & Disease Research, Laboratory of Neurobiology, Leuven, Belgium (A.d.B., A.S., L.R., L.P., W.R., R.L.).
³ University Hospitals Leuven, Department of Neurology, Belgium (W.R., R.L.).

PMID: 30852969
DOI: 10.1161/STROKEAHA.118.023857

No abstract available

Keywords: ephrins; gliosis; recovery; regeneration; stroke.

Publication types

Research Support, Non-U.S. Gov't

MeSH terms

Animals
Cerebral Cortex / metabolism*
Ephrin-A5 / genetics
Ephrin-A5 / metabolism*
Mice
Mice, Knockout
Neurons / metabolism
Recovery of Function / physiology*
Stroke / genetics
Stroke / metabolism*

Substances

Ephrin-A5