Platelet derived FasL contributes to apoptosis in stroke

Thromb Haemost. 2016 Oct 28;116(5):998-1000. doi: 10.1160/TH16-06-0447. Epub 2016 Aug 25.

Authors

Peter Kraft, Rebecca Schleicher, Marcus Olbrich, Michael K Schuhmann, Kelly Daryll Blanz, Frederic Emschermann, Sonja Ebenhoech, Christopher Starz, Ingo Hilgendorf, Sven G Meuth, Frank Edlich, Christoph Kleinschnitz¹, Harald F Langer²

Affiliations

¹ Christoph Kleinschnitz, MD, Department of Neurology, University Clinic Essen, Hufelandstr 55, 45147 Essen, Germany, Tel.: +49 201 723 6302, Fax: +49 201 723 5655, E-mail: neurologie@uk-essen.de.
² Harald F. Langer, MD, Section for Cardioimmunology, Eberhard Karls University, Otfried-Mueller-Straße 10, 72076 Tuebingen, Germany, Tel.: +49 7071 2982887, Fax: +49 7071 294474, E-mail: harald.langer@med.uni-tuebingen.de.

PMID: 27608763
DOI: 10.1160/TH16-06-0447

No abstract available

Publication types

Letter
Research Support, Non-U.S. Gov't

MeSH terms

Animals
Apoptosis*
Blood Platelets / metabolism*
Fas Ligand Protein / metabolism*
Humans
Inflammation
Jurkat Cells
Mice
Mice, Inbred C57BL
Platelet Activation
Stroke / metabolism
Stroke / physiopathology*
Thrombosis / metabolism

Substances

FASLG protein, human
Fas Ligand Protein
Fasl protein, mouse