Linking superinhibitory PLN mutations to CaMKII activation: a new arrhythmogenic mechanism in genetic DCM?

Cardiovasc Res. 2015 Jul 1;107(1):5-6. doi: 10.1093/cvr/cvv163. Epub 2015 May 26.

Authors

Michael Wagner¹, Silvio Weber², Ali El-Armouche¹

Affiliations

¹ Department of Pharmacology and Toxicology, TU Dresden, Fetscherstraße. 74, Dresden 01307, Germany michael_wagner@tu-dresden.de ali.el-armouche@tu-dresden.de.
² Department of Pharmacology and Toxicology, TU Dresden, Fetscherstraße. 74, Dresden 01307, Germany.

PMID: 26014576
DOI: 10.1093/cvr/cvv163

No abstract available

Publication types

Editorial
Comment

MeSH terms

Animals
Arrhythmias, Cardiac / etiology*
Calcium / metabolism*
Calcium-Binding Proteins / genetics*
Humans
Male
Mutation*

Substances

Calcium-Binding Proteins
Calcium