Synergistic suppression of Poly(I:C)-induced CCL3 by a corticosteroid and a long acting β2 agonist in nasal epithelial cells

Allergol Int. 2015 Apr;64(2):196-9. doi: 10.1016/j.alit.2014.10.006. Epub 2015 Feb 11.
No abstract available

Publication types

  • Letter
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adrenal Cortex Hormones / pharmacology*
  • Adrenergic beta-2 Receptor Agonists / pharmacology*
  • Cell Line
  • Chemokine CCL3 / antagonists & inhibitors*
  • Chemokine CCL3 / genetics
  • Chemokine CCL3 / metabolism
  • Dexamethasone / pharmacology*
  • Drug Synergism
  • Epithelial Cells / drug effects*
  • Epithelial Cells / metabolism
  • Formoterol Fumarate / pharmacology
  • Humans
  • Nasal Mucosa / cytology
  • Poly I-C / pharmacology*
  • RNA, Messenger / metabolism
  • Salmeterol Xinafoate / pharmacology

Substances

  • Adrenal Cortex Hormones
  • Adrenergic beta-2 Receptor Agonists
  • CCL3 protein, human
  • Chemokine CCL3
  • RNA, Messenger
  • Salmeterol Xinafoate
  • Dexamethasone
  • Poly I-C
  • Formoterol Fumarate