[c-MYC, PARP1 and BIN1 as targets for therapy of cancer cell resistance]

Med Sci (Paris). 2013 Feb;29(2):133-5. doi: 10.1051/medsci/2013292006. Epub 2013 Feb 28.
[Article in French]
No abstract available

Publication types

  • News

MeSH terms

  • Adaptor Proteins, Signal Transducing / genetics
  • Adaptor Proteins, Signal Transducing / physiology*
  • DNA Damage / genetics
  • Drug Resistance, Neoplasm* / genetics
  • Drug Resistance, Neoplasm* / physiology
  • Gene Expression Regulation, Neoplastic
  • Genes, Tumor Suppressor / physiology
  • Humans
  • Models, Biological
  • Molecular Targeted Therapy / methods*
  • Neoplasms / genetics
  • Neoplasms / therapy*
  • Nuclear Proteins / genetics
  • Nuclear Proteins / physiology*
  • Poly (ADP-Ribose) Polymerase-1
  • Poly(ADP-ribose) Polymerases / genetics
  • Poly(ADP-ribose) Polymerases / physiology*
  • Proto-Oncogene Proteins c-myc / genetics
  • Proto-Oncogene Proteins c-myc / physiology*
  • Tumor Suppressor Proteins / genetics
  • Tumor Suppressor Proteins / physiology*

Substances

  • Adaptor Proteins, Signal Transducing
  • BIN1 protein, human
  • MYC protein, human
  • Nuclear Proteins
  • Proto-Oncogene Proteins c-myc
  • Tumor Suppressor Proteins
  • PARP1 protein, human
  • Poly (ADP-Ribose) Polymerase-1
  • Poly(ADP-ribose) Polymerases