Herpes zoster optic neuritis

Int Ophthalmol. 2011 Jun;31(3):233-6. doi: 10.1007/s10792-011-9443-y. Epub 2011 May 28.

Abstract

Herpes zoster (HZ) is an acute infection caused by reactivation of the latent varicella-zoster virus [1]. Herpes zoster ophthalmicus (HZO) occurs when inflammation spreads from the ganglion of Gasser to the ophthalmic branch of the trigeminal nerve. Optic neuritis, a very rare sequela of HZO [2-4], can occur simultaneously to the acute vesicular skin eruption or, more frequently, as a postherpetic complication. We report on a 74-year-old woman who presented with HZ optic neuritis 45 days after developing an incompletely treated bout of trigeminal HZ, characterized only by pruritus. It is important to value the non-specific manifestations of cutaneous HZ in the prodromal phase, so as to offer timely and appropriate treatment.

Publication types

  • Case Reports

MeSH terms

  • Acyclovir / administration & dosage
  • Aged
  • Antiviral Agents / administration & dosage
  • Diagnosis, Differential
  • Evoked Potentials, Visual
  • Female
  • Fluorescein Angiography
  • Follow-Up Studies
  • Fundus Oculi
  • Herpes Zoster Ophthalmicus / diagnosis*
  • Herpes Zoster Ophthalmicus / drug therapy
  • Herpes Zoster Ophthalmicus / physiopathology
  • Humans
  • Injections, Intravenous
  • Optic Nerve / pathology*
  • Optic Neuritis / diagnosis*
  • Optic Neuritis / drug therapy
  • Optic Neuritis / virology
  • Visual Acuity

Substances

  • Antiviral Agents
  • Acyclovir