Repeated sprint swimming performance after low- or high-intensity active and passive recoveries

J Strength Cond Res. 2011 Jan;25(1):109-16. doi: 10.1519/JSC.0b013e3181b22a9a.

Abstract

The purpose of this study was to examine the effects on sprint swimming performance after low- and high-intensity active recovery (AR) as compared to passive recovery. Ten male competitive swimmers (age: 17.9 ± 2.3 years; body mass: 73.2 ± 4.0 kg; height: 1.81 ± 0.04 m, 100-m best time: 54.90 ± 1.96 seconds) performed 8 × 25-m sprints with 120-second rest intervals followed by a 50-m sprint 6 minutes later. During the 120-second and the 6-minute interval periods swimmers rested passively (PAS) or swam at an intensity of 40% (ACT40; 36 ± 8% of the V(O2)max) and 60% (ACT60; 59 ± 7% of the V(O2)max) of their individual 100-m velocity. Performance time of the 8 × 25-m after ACT60 was slower compared with PAS and ACT40, but no difference was observed between ACT40 and PAS conditions (PAS: 12.15 ± 0.48, ACT40: 12.23 ± 0.54, ACT60: 12.35 ± 0.57 seconds, p < 0.05). Performance time of the 50-m sprint was no different between conditions (PAS: 26.45 ± 0.91; ACT40: 26.30 ± 1.18; ACT60: 26.21 ± 1.19 seconds; p > 0.05). Blood lactate concentration was not different between PAS, ACT40, and ACT60 after the 8 × 25-m and the 50-m sprints (p > 0.05). Passive recovery, or low intensity of AR (40% of the 100-m velocity), is advised to maintain repeated 25-m sprint swimming performance when a 2-minute interval period is provided. Active recovery at an intensity corresponding to 60% of the 100-m velocity decreases performance during the 25-m repeated sprints without affecting the performance time on a subsequent longer duration sprint (i.e., 50 m).

MeSH terms

  • Adolescent
  • Athletes
  • Athletic Performance / physiology*
  • Humans
  • Lactic Acid / blood
  • Male
  • Muscle Fatigue / physiology
  • Oxygen Consumption / physiology
  • Recovery of Function
  • Swimming / physiology*
  • Young Adult

Substances

  • Lactic Acid