Failure to confirm association between SLC22A4 polymorphism and rheumatoid arthritis in a Japanese population

Arthritis Rheum. 2005 Sep;52(9):2947-8. doi: 10.1002/art.21244.

Authors

Mahito Kuwahara¹, Katsunori Ikari, Shigeki Momohara, Takahiro Nakamura, Masako Hara, Hisashi Yamanaka, Taisuke Tomatsu, Naoyuki Kamatani

Affiliation

¹ Institute of Rheumatology, Tokyo Women's Medical University, Tokyo, Japan.

PMID: 16142713
DOI: 10.1002/art.21244

No abstract available

Publication types

Research Support, Non-U.S. Gov't

MeSH terms

Arthritis, Rheumatoid / diagnosis
Arthritis, Rheumatoid / epidemiology
Arthritis, Rheumatoid / genetics*
Cohort Studies
Core Binding Factor Alpha 2 Subunit
DNA-Binding Proteins / genetics
Female
Genetic Predisposition to Disease*
Genotype
Humans
Japan / epidemiology
Linkage Disequilibrium / genetics
Male
Organic Cation Transport Proteins / genetics*
Polymorphism, Genetic*
Proto-Oncogene Proteins / genetics
Symporters
Transcription Factors / genetics

Substances

Core Binding Factor Alpha 2 Subunit
DNA-Binding Proteins
Organic Cation Transport Proteins
Proto-Oncogene Proteins
RUNX1 protein, human
SLC22A4 protein, human
Symporters
Transcription Factors