Over-expression of rexA nullifies T4rII exclusion in Escherichia coli K(lambda) lysogens

Can J Microbiol. 2004 Feb;50(2):133-6. doi: 10.1139/w03-115.

Abstract

Dosage and relative cellular levels of RexA and RexB proteins encoded by the rexA-rexB genes of a lambda prophage are important for the Rex+ phenotype, which was nullified when greater RexA or RexB was provided than was necessary for the complementation of a rexA- or a rexB- prophage.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Bacteriophage T4 / genetics*
  • Bacteriophage T4 / growth & development
  • Bacteriophage lambda / genetics*
  • Bacteriophage lambda / physiology
  • Escherichia coli / virology*
  • Gene Deletion
  • Gene Expression
  • Genes, Viral / genetics
  • Genes, Viral / physiology
  • Genetic Complementation Test
  • Lysogeny*
  • Mutation
  • Plasmids
  • Prophages / genetics
  • Prophages / physiology
  • Viral Nonstructural Proteins / genetics*
  • Viral Proteins / genetics*

Substances

  • RexA protein, Bacteriophage lambda
  • Viral Nonstructural Proteins
  • Viral Proteins
  • rIIA protein, Enterobacteria phage T4
  • rexB protein, Bacteriophage lambda