Transforming growth factor-beta: a local or systemic mediator of plaque stability?

Circ Res. 2001 Nov 9;89(10):853-5.
No abstract available

Publication types

  • Editorial
  • Research Support, Non-U.S. Gov't
  • Comment

MeSH terms

  • Animals
  • Apolipoproteins E / deficiency
  • Apolipoproteins E / genetics
  • Arteriosclerosis / complications
  • Arteriosclerosis / immunology
  • Arteriosclerosis / physiopathology*
  • CD40 Antigens / metabolism
  • CD40 Ligand / metabolism
  • Disease Models, Animal
  • Disease Progression
  • Humans
  • Immunoglobulin Fc Fragments / genetics
  • Inflammation / complications
  • Inflammation / immunology
  • Inflammation / physiopathology
  • Macrophages / immunology
  • Mice
  • Mice, Knockout
  • Protein Serine-Threonine Kinases
  • Receptor, Transforming Growth Factor-beta Type II
  • Receptors, Transforming Growth Factor beta / administration & dosage
  • Receptors, Transforming Growth Factor beta / genetics
  • Recombinant Fusion Proteins / administration & dosage
  • Recombinant Fusion Proteins / genetics
  • Signal Transduction / drug effects
  • T-Lymphocytes / immunology
  • Transforming Growth Factor beta / antagonists & inhibitors
  • Transforming Growth Factor beta / genetics
  • Transforming Growth Factor beta / metabolism*

Substances

  • Apolipoproteins E
  • CD40 Antigens
  • Immunoglobulin Fc Fragments
  • Receptors, Transforming Growth Factor beta
  • Recombinant Fusion Proteins
  • Transforming Growth Factor beta
  • CD40 Ligand
  • Protein Serine-Threonine Kinases
  • Receptor, Transforming Growth Factor-beta Type II